GASTROENTEROPATHY, PROTEIN LOSING

Gastroenteropathy, protein losing, is defined by an abnormally high loss of proteins in the gastrointestinal system, leading to edema, hypoproteinemia, and, in certain instances, pericardial effusions and pleural. Furthermore, gastroenteropathy, protein losing must be examined in individuals with hypoproteinemia who have ruled out other reasons, including severe proteinuria, malnutrition, and reduced protein synthesis because of liver illness.

SYMPTOMS

Symptoms of gastroenteropathy, protein losing are the following:

  • Indigestion
  • Hypoproteinemia
  • Lower limb edema
  • Impaired Immune system

DIAGNOSIS

To diagnose gastroenteropathy, protein losing, the doctor may perform the following:

  • Ascites Testing. This procedure includes checking ascites cells, chylous microparticles, proteins, enzymes, and malignant cells.
  • Lymphangiography. This method may assist in differentiating between congenital and subsequent intestinal lymphangiectasia. Intestinal lymphangiectasia is an uncommon digestive condition defined by abnormally expanded lymph veins feeding the small intestinal lining.
  • Jejunal Mucosa Biopsy. In this procedure, a section of the upper small intestine’s lining (mucosa) is removed. This may be done surgically, but it is typically done using an enteroscope or, less often, with a specific metal capsule (Crosby capsule) consumed by the patient.

TREATMENT

Gastroenteropathy, protein losing has various effective treatment strategies depending on the causes. Symptomatic therapy, such as diuretics, a low-salt diet, and intravenous injections of human albumin, has only a short-term impact. 

  • Supportive Therapy for Symptoms. Diuretics may be used to replenish human serum albumin in patients with serous cavity effusion or edema caused by hypoproteinemia; pancreatin preparation and vitamins can be administered to supplement patients with fatty diarrhea and vitamin deficiencies.
  • Diet. Patients with severe edema should be provided a high-calorie diet and high protein. In contrast, those with mild edema must be given a low-fat or medium-chain triglyceride (MCT) diet to lessen the burden of intestinal lymphatic capillaries.
  • Hypoproteinemia Correction. Intravenous injection of natural albumin has only a transient impact. In general, it is not recommended to address hypoproteinemia alone by administering human albumin but rather to improve plasma protein content through etiological therapy and dietary changes.
  • Diuretics. Potassium-expelling diuretic and potassium-preserving, such as thiazides and spiral lactones, may be administered in combination. Furosemide potent diuretics can be administered as required to minimize edema and ascites.

Antibiotics. Vitamin deficiencies should be treated with vitamins, while calcium and magnesium deficiencies should be treated with medicines.

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